Teeth sensitivity – Prevention and Management

Teeth sensitivity, is a very commonly encountered dental condition. The discomfort and pain caused by this condition, actually, makes consumption of hot, cold and sweet food difficult to relish. A right diagnosis and well planned treatment can keep tooth sensitivity at bay.


SYMPTOMS

Teeth become sensitive to temperature changes. most commonly teeth respond to cold food.

If the teeth elicits a pain after a hot stimulus, then it means that, the nerve of that tooth is damaged, and it will need a root canal therapy.

If the pain in the tooth lingers on for several minutes, even after removal of the stimulus, its a sign of pulpal damage, and will need root canal therapy.

sometimes, immediate post-filling, the patient complains of that tooth being sensitive. this is mos commonly seen with silver and composite fillings. the silver in the amalgam restorative material transmits the cold to the nerves. in case of bonded restorations, etching of the tooth surface is done, before placing, and curing the restorative material. This acid etching also leads to teeth sensitivity. But with the entry of the new generation of bonded restorations, this complication can well be avoided.


DIAGNOSIS

The dentist will ask the for the following details,

A detailed, teeth cleaning methods will be asked – an improper brushing technique, along with a hard bristle, tooth brush and an abrasive dentifrice leads to tooth wear and teeth sensitivity.

Dental history – to assess the motivation levels of the patient. presence of long standing deposits, like plaque and calculus can also make the tooth sensitive.

Pulp testing will be done, to check whether the tooth is vital or not. In case of a non vital tooth, which means that the nerve is completely damaged, a root canal treatment is compulsory.

Dietary habits of the patient – consumption of acidic and more citrus food, alcoholic beverages.

teeth examination for signs of decay, gum recession, gum disease, chipped filling.


MANAGEMENT

Teaching the patient the right brushing technique. Giving a demonstration really helps the patient to understand, and emulate the dentist.

If the sensitivity is due to pulpal damage, a root canal therapy puts an end to the agony.

Sensitivity after an amalgam filling, vanishes in few days. So it does not require any further measures.

A high filling restoration can be altered by polishing it.

Tooth pastes that are made exclusively for sensitive teeth can be used, fluoride varnishes are also available. They act by blocking the dentinal tubules. This eradicates pain sensation.

Avoiding acidic products for oral hygiene purposes, for example few flouride rinses are acidic y nature.

Consult a dental professional for periodic oral prophylaxis.



Sensitive Teeth Symptoms & Causes (Dentine Hypersensitivity)

Sensitive Teeth, also called, Dentine hypersensitivity has been accepted to be among the most painful dental conditions, affecting oral comfort and function.

Earlier, dentine hypersensitivity was considered to be an enigma, since it was frequently encountered, but poorly understood.

Dentine hypersensitivity is defined as, ” a condition, characterized by short, sharp pain arising from exposed dentine in response to stimuli typically thermal, evaporative, tactile, osmotic, or chemical and which cannot be ascribed to any other form of dental defect or pathology.”


TERMINOLOGIES


Dentine hypersensitivity is also referred to as,

Dentine sensitivity

Dentinal hypersensitivity

Cervical hypersensitivity

Root hypersensitivity

Cemental hypersensititvity


Causes of Sensitive Teeth or Dentine Hypersenstivity


  1. Caries
  2. Chipped tooth
  3. Fractured restorations
  4. Marginal leakage around restorations
  5. Some restorative materials
  6. Cracked tooth syndrome
  7. Palatogingival grooves
  8. Gingival recession, with rapid loss of cementum/loss of enamel
  9. Openeing of dentinal tubules due to either, attrition, abfraction, abrasion, erosion.
  10. Parafunctional habits such as bruxism, causes occludsal hypersensitivity
  11. Chewing on coarse diets, or abrasive materials
  12. Vigorous tooth brushing with an abrasive dentifrice
  13. High consumption of citric diet, like, oranges and lemon.
  14. Acidity
  15. Excess intake of alcoholic beverages, soft drinks, yoghurt.


MECHANISMS OF DENTINE SENSITIVITY


The Hydrodynamic theory is the most accepted, which explains the mechanism of dentine hypersensitvity.

according to this theory,


Most pain-evoking stimuli increases the outward flow of the fluid within the dentinal tubules

This fluid movement, inturn, causes a pressure change

This activates the A- delta   intradental nerves, at the pulp dentine border; or within the dentinal tubules.


Pain is elicited.



CONCLUSION

Sensitive Teeth or Dentine hypersensitivity, in terms of symptoms, and localisation of lesions, has all the hallmarks of tooth wear phenomenon.

A great deal still needs to be researched.


Diet And Dental Caries

Complete eradication of dental caries which is one of the most infectious diseases know to man, is still a distant dream.

We know that 3 main factors: host tooth, microbes and diet cause tooth decay.

A lot of research has been carried out in order to assess the effect of diet on dental decay.

ROLE OF DIET IN THE CARIES PROCESS

Caries development probably does not become severe unless the consumption of ferment-able carbohydrates is excessive.


Product related dietary factors

Type of diet

Natural sugars as such potential to cause decay as sugar. the more fibrous nature of food consumed, lesser are the chances of tooth decay.

Retention time of dietary components

Sugars stick on to the surface of the teeth and increase the risk of dental deacay. The greater amount of time sugars remain in contact with the tooth surface, greater the decay.

Protective factors-

Caries incidents is not only related to the cariogenesity of the diet but also to the protective effects of the saliva, cariostatic components of the food, etc

Intake frequency and oral clearance time

Greater the intake and consumption of the cariogenic food, greater the susceptibility to deacy.

Longer the oral clearance time, greater the decay.

See my previous post for a complete list of Dental caries causes, symptoms,diagnosis and treatment.

PROTECTIVE FOOD COMPONENTS

Some food components are somewhat caries inhibiting and have been called protective especially if its high in calcium and phosphates.

They act by,

  • Reducing the rate of dissolution of hydroxyapatite.
  • Reducing the fall in plaque pH by buffering acids produced by fermentation.
  • Enhancing remineralization
  • Modifying formation and composition of pellicle and plaque.

Other cariostatic dietary components include peanuts, cocoa, lectin, cheese.


Fats are also known to reduce the cariogenesity of foods by

  • Replacement of carbohydrates,
  • Formation of a protective barrier on the enamel
  • Certain fatty acids have an anti microbial effect and thus they inhibit glycolysis in human dental plaque.

SUGAR SUBSTITUTES

These are also referred to as caloric or nutritive or carbohydrate sugar substitute.

The most important group is the sugar alcohols also called glycitols.

DIETARY ADVICE


This is the most important aspect of dental caries prevention.

  • The individual compliance with the clinicians advice is very important.
  • If the patients do not co-operate the result is bound to be un productive.
  • The following scheme states the course of action-
  • Thorough examination including dental and medical history.
  • Evaluation of probable causes
  • Starting patient counseling regarding diet
  • Evaluating patient’s compliance
  • Determination of regular eating habits
  • Continuation of dietary advice
  • Giving the patient a personalized dietary program.
  • The record of the dietary intake should be maintained by the patient.
  • This record will include all details regarding the type, quantity and the time when the food was consumed.

SUMMARY OF DIETARY MEASURES FOR PREVENTION AND CONTROL OF DENTAL CARIES

Frequency of meal- No. of meals plus snacks should be kept on low levels.

Amount and concentration of sucrose in meal- A low sugar consumption is desirable.

Estimation of sugar- Sugar should be eliminated as fast as possible from the oral cavity.

Consistency of food- Food needing active chewing leads to an increased salivation which is desirable.

Fermentable carbohydrates- Polysaccharides,  Di-saccharides and Mono-saccharides can contribute to acid formation in the oral cavity, but the capacity differs between products.

Sugar substitute- Lowers acid formation.

Protective and favorable element in diet- Fluoride in food or drinking water, phosphate, calcium, fat and protein also possess caries inhibitory properties.

CONCLUSION

Changing dietary habits to improve health is not easy. it is important to realize that dietary habits do change and it is the duty of the health professional to encourage them to change in the right direction.

Recent Advances in Dental Composite

The concept of the aesthetic dentistry and tooth coloured restorations are not new. Though the aesthetic restorations are in vogue, today and their demand is increasing day by day, the translucent silicates, were even available  in late 19th century and early 20th century.

Composite Filling Material

COMPOSITION

The components of composite resin are,

  • Resin matrix
  • Fillers
  • Coupling agents
  • Coloring agents

ADVANCES IN COMPOSITES

1. Flowable Composites

Introduced in 1996, the flowable composites are characterized by the presence of filler particles that have a particle size similar to that of the traditional hybrid composites, but the filler content is reduced which results in a decrease in viscosity.

Flowable composites were launched to improve upon the handling characteristics of existing composites.

Features

  • Because of the lesser amount of filler loading, the flow is increased and the material flows immediately on dispensing from the syringe/compule.
  • The depth of cure is approximately 6mm.
  • Mechanical properties of the material, like compressive strength, diametrical tensile strength, biaxial flexure strength, are generally about 60-90% of those of  the conventional composites. Hence the use of these materials should be avoided in high stress areas.
  • The low contact angle associated with the flowable composite resins is an indication of how well it will wet and relate to all of the irregularities in the cavity preparation.
  • When using the flowable composite resin, it is important to cover all the dentin.
  • If any space exists between the matrix band and the gingival margin, there is the potential for allowing the flowable to seep beyond the gingival margin, thereby creating an overhang, which is difficult to remove.
  • The thickness should range between 0.5-1micron. Thickness greater than 1mm, is acceptable. However, it is important not to cover the occlusal margins or establish the proximal surface with the flowable agent.


Application areas for Flowable Composites –

  • Filling materials in low stress areas.
  • Useful in areas of difficult access, like in repairing amalgam, composite or crown margins, pit and fissure sealing, as liners in proximal boxes of class II,CLASS III, and class V cavities.
  • Repairing porcelain
  • Tunnel restorations
  • Core build up
  • Veneer material
  • Cementing agents for porcelain restorations.

Inferior characteristics include,

  • Curing shrinkage
  • Water sorption
  • Reduced compressive strength
  • Low elastic modulus
  • Increased wear resistance


Functions

Development of an intimate relationship between the walls of the cavity preparation and the surface of the flowable resin.

It serves as a stretchable liner to absorb the shrinkage or contraction of the overlying composite resin restoration.


Before purchase, points to consider,

  • Its handling characteristics
  • Radioopacity
  • Cost


Packable Composites

A major change in the resin viscosity came about in 1998, with the first introduction of the so called packable composite resin. it is based on the newly introduced concept PRIMM. (Polymer rigid inorganic matrix material). This concept was introduced to help the clinician switch over from amalgam to composite resins. It was believed that if, the composite resin exhibited the handling characteristics of amalgam, the clinician would more readily accept the new material. this system consists of a resin and a ceramic component.

It should be pointed out, that some practitioners refer to the new composite resins as condensable. This is different from packable.


Improved properties over conventional composites,

  • Increased flexure modulus
  • Increased resistance to wear
  • Higher depth of cure.
  • Reduced polymerization shrinkage.
  • Non stickiness to the instrument.
  • Antibacterial composites

Composites that offer Antibacterial properties

Are promising since, several studies have shown that a greater amount of bacteria and plaque accumulate on the surface of the resin composites than on the surface of other restorative materials/enamel surface. Therefore, attempts to incorporate antibacterial properties into resin composites are a welcome step.

Expanding Matrix resin for composites

Composite resins that expand slightly during polymerization are highly desirable as these would facilitate bulk placement of the material and reduce post operative sensitivity.

Laser curing of composite resins

Recently composite resins are bieng cured with lasers. argon laser is the most suited. the intensity of laser required is 250mW, and the time required is 10 seconds per increment.

CONCLUSION

Today, the combination of highly wear resistant composite resins and the ability to bond to dentin has lead to the potential for total replacement of amalgam with a more esthetic material.

The composite resin used by the finishing today represents the accumulation of years of research, and clinical testing. the products of tomorrow, promise to be even more exciting.


The Flip Side of Teeth Whitening Procedures

Teeth whitening, also called Dental Bleaching, is a very common and a well known procedure in the field of dentistry. This aesthetic procedure has become increasing popular in the recent years.

With increasing age, the adult teeth enamel become less porous, due to which they take up a darker hue. Teeth also become stained by food pigments, tobacco, bacterial products. Certain medications like tetracycline are also known to cause tooth discoloration.

They are a range of methods used to brighten teeth, namely, bleaching gels, bleaching strips, bleaching pens, natural bleaching, laser bleaching.

The procedure can be carried out at home, by the individual himself, or in a dental office by a dentist, which is recommended.

The traditional home whitening involves applying bleaching gel to the teeth using thin guard trays. It can also be done using small bleaching strips which are applied over the front surface of the teeth.

In  a dental office, Power bleaching is performed by the professional, which is usage of light energy to accelerate the process of bleaching.


HOW DOES IT REALLY WORK ?

Bleaching products, use oxidizing agents like Hydrogen  Peroxide or Carbamide peroxide to lighten the tooth shade.

The tooth enamel naturally presents with a rod-like crystal structure, which have porosities.

The oxidizing agent penetrates these porosities, and oxidizes the interprismatic stain deposits.

Over a period of time, the underlying dentin layer also gets bleached.

The effects of bleaching lasts for several months, which depends on the individual’s pattern of living,and habits.

Habits like smoking,usage of any form of tobacco, drinking excessive amounts of dark colored liquids like tea, coffee, red wine limits the bleaching effect.

Side Effects of Teeth Whitening or Dental Bleaching


All good things have their share of shortcomings, and Dental Bleaching procedure is no exception. Though it has been a breath of fresh air, and a perfect solution to the numerous issues encountered in cosmetic dentistry, it sure does have a flip side to it.

  • The most common side effect of bleaching is increased tooth sensitivity, which also leads to pain. This is mainly caused due to open dentinal tubules. The patient experiences pain on eating hot,cold, or sweet food products. This is commonly seen in the early stages of bleaching treatment. These are temporary and reversible, and disappear within 3 days of stopping or treatment completion.


  • Mild irritation of soft tissues in the mouth especially the gums. This too is reversible.


  • Some over-the-counter bleaching products contain carbamide peroxide, most of them are Hydrogen Peroxide based, which has the potential to interfere with DNA synthesis.


  • If proper precautions are not taken, and the high concentration oxidizing agent comes in contact with the soft tissues, it can cause severe chemical burns. It also has the potential to bleach the mucous membrane.


  • White…whiter…whitest – Over-white teeth, which results from excess bleaching. Also called Hyperodonto-oxidation.


  • Very intense bleaching treatment, results in large change in tooth shade, in less than an hour, but they carry the risk of reversing the effect. The tooth appears exactly the way it was in the start.


  • Pregnant and lactating women are contraindicated for Tooth whitening procedure. Accidental swallowing of the bleaching gel can affect the unborn child’s dentition to some extent.


  • Many individuals resort to several home remedies to whiten their teeth. They usually rely on the acidic constituents, which have the ability to easily erode the hardest of mineralized tooth structure over a period of time.


  • Teeth whitening procedures are not advised in some cases no matter how discolored the teeth may be. For instance, children below 16 years of age.


  • Individuals with defective restorations, receding gums have to cautious before the bleaching procedure.


  • Individuals allergic to peroxide (whitening agent) should refrain from using bleaching products.


  • Any gum diseases, or dental caries if present must be treated prior to any teeth whitening procedures. The bleaching product can seep into the existing cavities and enter the inner areas of the tooth and eventually cause sensitivity and pain.

Tooth Avulsion – It’s a knock out!

The Avulsed or Luxated tooth is both a dental and an emotional problem. It is usually the result of trauma to an anterior tooth of a child or young adult. the shock and pain of the injury and the loss of a tooth needed for eating, speaking and smiling, often lead to emotional upheaval in patient and parent.

The longer the luxated tooth is out of the socket, the less likely it will remain in a healthy, functional state after replantation.

The extraoral time for an avulsed tooth optimally should not exceed 30 min, the patient must be taken to the dentist immediately.


MANAGEMENT

Can be divided into,

a. Management at the site of injury.

b. Management in the dental office.


a. Management at the site of injury:

  • Wash the tooth in running water without brushing or cleaning it, and examine it to be certain that the tooth is intact.
  • Have the patient rinse mouth. Replace tooth in its socket using gentle, steady finger pressure.
  • If the patient is cooperative and able, have the patient gently close the teeth together to force the teeth together to force the tooth back into its original position.
  • If immediate replantation is not possible, place the tooth in the best transport medium available.

Transport Media –

  • Hank’s Balanced Salt Solution (H.B.B.S)
  • Milk
  • Saline
  • Saliva (buccal vestibule)
  • If none of these is readily available, use water.


b. Management in the dental office:

i. Replantation of Tooth –

  • If extraoral dry time is more than 2 hours, replant immediately.
  • If it exceeds 2 hours, soak the tooth in a topical Fluoride solution for 5-20 minutes, rinse in saline and relant.
  • If tooth has been in any of the physiologic transport medium, then it is safe to replant immediately.


ii. Management of the Root surface -

  • Keep the tooh moist at all times.
  • Do not handle the root at all. (hold the tooth by its crown portion)
  • Do not scrape or brush the root surface, or attempt to remove the root tip.
  • If the root appears clean, replant as it is after rinsing with saline.
  • If its contaminated, rinse with H.B.B.S or saline (use tap water if none of these are available).
  • In case of persistent debris, gently use cotton pellet or a wet sponge to remove the remaining debris.


iii. Management of the socket -

  • Gently aspirate without entering the socket, if a clot is present, use light irrigation with saline.
  • Do not curette the socket.
  • Do not vent the socket.
  • Do not make a surgical flap unless bony fragments prevent replantation.
  • If the alveolar bone is collapsed, and prevents replantation, carefully insert a blunt instrument into the socket to reposition the bone to its original position. Aafter replantation, manually compress facial and lingual bony plates.


iv. Management of Soft tissue -

  • Tightly suture any soft tissue lacerations.


Splinting

  • Use acid etch resin alone or with soft arch wire, or useorthodontic brackets with passive arch wire.
  • Splint should remain in place for 7-10  days; however if tooth demonstartes excessive mobility, splint should be replaced until mobility is within acceptable limits.
  • Bony fractures resulting in mobility usually require longer splinting periods (2-8 weeks).

Precautions,

  1. No biting on splinted teeth. The teeth should be kept out of occlusion.
  2. Soft diet
  3. Good oral hygiene.

Adjunctive Drug therapy

  • Systemic Antibiotics
  • Aanalgesics
  • Referral to physician for tetanus consultation within 48 hours.
  • Chlorhexidine rinses.


Endodontic therapy


i. Tooth with open apex (divergent roots) and less than 2 hours extraoral dry time:

  • Replant in an attempt to revitalize the pulp.
  • Recall patient every 3-4 weeks for evidence of pathosis.
  • If pathosis is noted thoroughly clean and fill the canal with Calcium Hydroxide (apexification).


ii. Tooth with open apex, and more than 2 hours extraoral dry time:

  • Thoroughly dry the canal and fill it with Calcium hydroxide.
  • Recall the patient every 6-8 weeks.


iii. Tooth with partially to completely closed apex and less than 2 hours extraoral dry time:

  • Pulp extirpation in 7-14 days.
  • Medicate the canal with Calcium hydroxide.
  • Obturate canal with Gutta Percha points and a sealant after 7-14 days of calcium  hydroxide.


iv. Tooth with partially or completely closed apex, and greater than 2 hours of extraoral time:

  • Perform root anal therapy intraorally or extraorally.
  • If treated extraorally, avoid chemical or mechanical damage to root surface.
  • Avulsed permanent teeth requires follow up evaluations for a minimum of 2-3 years to determine the outcome of therapy.


COMPLICATIONS  OF  TOOTH  REPLANTATION

  1. Inflammatory resorption
  2. Replacement resorption
  3. Ankylosis
  4. Tooth Submergence

Dental Caries – Another reason to kick your sugar habit

It is very strange that the hardest tissue of the body – the enamel, which is indestructible otherwise, can disintegrate in the oral environment. Caries (Latin meaning ‘dry rot’) is the name given to the prcess of slow disintegration that may affect any of the biologic tissues by bacterial action. Usually, such disintegration also affects the tooth, that is why the term ‘Dental Caries’ is common. In a simple manner, Dental Caries can be defined as ‘the irreversible, slow progressing decay of hard tissues of the tooth’.

Dental Caries













CLASSIFICATION

Dental caries can be classified according to 3 major factors:

a. According to tooth Morphology –

  • Pit and Fissure Caries
  • Smooth Surface Caries
  • Root Caries
  • Linear Enamel Caries

b. According to severity and progress of the lesion –

  • Incipient Caries
  • Rampant Caries
  • Arrested Caries
  • Recurrent Caries
  • Radiation Caries

c. According to Age pattern –

  • Nursing bottle Caries
  • Adolescent Caries
  • Geriatric Caries.


ETIOLOGY OF DENTAL CARIES –

Dental Caries is a multifactorial disease. There are 4 factors contributing to the caries process, which is explained below,


1. Host Factor

a. Tooth Factor

  • Morphology and position in the arch
  • Chemical nature

b. Saliva

  • Composition, pH, antibacterial activity
  • Quantity and viscosity of salivary flow

2. The Microflora

3. The substrate/diet

  • Physical nature
  • Chemical nature

4. Time


1. The Host Factor

a. Tooth

  • Morphology, and position in the arch –

Deep pits and fissures in any tooth make them susceptible to decay because of food impaction and bacterial stagnation. That is the reason for occlusal surfaces to be an easy target for decay. Irregularities in the arch form, crowding of the teeth also favors development of caries. Partially impacted third molars are more prone to caries and so are the buccally or lingually placed teeth.

  • Chemical nature –

Surface enamel is more caries resistant than subsurface enamel. The surface enamel has more minerals and less water content, compared to the deeper layers. In addition certain elements such as Fluoride, chloride, zinc lead etc. accumulates more on the enamel surface than the subsurface. With the passage of time, teeth become more resistant to caries because of decrease in permeability and increase in nitrogen and fluoride content.

b. Saliva

  • Composition, pH, antibacterial activity

Caries prone individuals have low calcium and phosphorous levels. Caries immune people have greater ammonia content in saliva, probably because the higher ammonia content in saliva retards plaque formation and neutralizes acid formation to a certain extent.

The pH at which saliva ceases to be saturated with calcium and phosphorous is called critical pH. Under normal conditions, the critical Ph is 5.5. Below this value, the inorganic content of the tooth may dissolve. A fall in the buffering capacity of saliva leads to increase in caries incidence.

Lysozyme, an antibacterial agent in saliva, can inhibit airborne and water borne organisms in the oral cavity to some extent. It is effective against cariogenuc micro organisms maintiaining the etiologic balance of oral flora.

  • Quantity and Viscosity of salivary flow –

Human beings suffering from decreased flow of saliva or lack of salivary secretions (Xerostomia) usually experience increased rate of tooth decay. This is evidenced in various diseases such as Sjogren’s syndrome, Sarcoidosis, Diabetes etc.

Under decreased salivary flow, it has been observed that there occurs an alteration in the microflora due to decreased salivary buffering capacity which favors the growth of more aciduric yeasts.

2. Microflora

Tooth decay cannot occur without the presence of micro organisms.

3. The substrate or diet

  • Physical nature of diet –

The diet of primitive man consisted of raw food including sand, soil coatings, which led to attrition and also cleansing the teeth off the debris, thereby reducing caries. Modern diet includes, refined foods, soft drinks, and eatables which lead to collection of debris predisposing to more caries.

  • Chemical nature of diet -

The main ingredient is carbohydrate, which is accepted as one of the most important factor in dental caries process. Basically, following factors are responsible as far as diet and dental caries is concerned,

i. Particle size or roughness of diet

ii. Palatability of diet

iii. Eating and drinking pattern; after diet and within diet

iv. Retention and clearance of diet

v. Age at which diet is offered.


SIGNS  AND  SYMPTOMS

An individual affected by the dental decay is usually unaware about it. The carious lesion starts, as a small chalky white spot on the enamel surface, which is asymptomatic. This early sign of enamel demineralization is called incipient decay. This eventually progress to a full blown cavity. once the cavitation takes place, the process becomes irreversible. A brown, shiny lesion indicates that dental caries was present there, which has undergone remineralization, which is also called arrested caries A dull appearance on the contrary suggests, an ongoing demineralization process.

Once the decay passes through enamel, the dentinal tubules, which have passages to the nerve of the tooth, become exposed and elicits pain. Fctors like heat, cold, sweet foods or drinks can aggravate the pain.


DIAGNOSIS

a. Visual Examination:

  • Includes, looking for cavitation, surface roughness, opacification and discoloration.
  • Teeth are cleaned and dried with compressed air, and viewed under adequate light source.

b. Tactile Examination:

Tip of the Dental Explorer used for diagnosis of Caries


  • Includes, determing roughness or softness of the tooth surface, with a sharp explorer.
  • Both, penetration and resistance of an explorer tip (catch) have been interpreted as an evidence of demineralization.


Various aids for diagnosis of caries include –

I. Radiographic methods:

  • Conventional Radiography
  • Xero Radiography
  • Digital Imaging
  • Computer Image Analysis
  • Subtraction Radiography

II. Electrical Resistance:

  • An instrument called ‘Van Guard Electronic Cries Detector’ has been designed to measure the electrical conductivity of the tooth.
  • The electrical conductivity is directly proportional to the maount of demineralization that has occured.
  • Electrical resistance is measuring the electrical conductivity through the pores.

III. Ultraviolent Illumination:

  • Ultraviolent light (UV) has been used to increase the optical contrast between the carious region and the surrounding sound tissue.

IV. Laser Auto Fluorescence (LAF)

V. Endoscope/Videoscope:

  • Endoscopic technique is based on observing the fluorescence that occurs when tooth is illuminated with blue light in the wavelength range of 400-500nm.
  • Difference is seen in the fluorescence of sound enamel and carious enamel.

VII. Ultrasonic imaging:

  • Ultrasonic imaging was introduced for detecting early  carious lesions in smooth surfaces. The demineralization of natural enamel is assessed by ultrasound pulse echo technique.

VIII. Dye Penetration Method.


TREATMENT

Topical fluoride application helps in remineralization of small incipient lesions. The goal of treatment is to preserve tooth structures and prevent further destruction of the tooth.

Some  Prescription medications—may be required in some cases to relieve pain during or following treatment or to relieve anxiety during treatment. In a cavity, large portions of the decayed material from the tooth is removed using the dental handpiece (drill). A spoon excavator is used, manually to scoop out the soft carious material, from the depth of the cavity. After complete removal of the decay, the tooth is ready for restoration. This helps the tooth to regain its function and aesthetic condition.

Restorative materials include dental amalgam, porcelain, composite resins, gold.  Composite resin and porcelain can be made to match the color of a patient’s natural teeth and are thus used more frequently when aesthetics are a concern. Composite restorations are not as strong as dental amalgam and gold; some dentists consider the latter as the only advisable restoration for posterior areas where chewing forces are great.

Amalgam Restoration


In case of extensive decay, there may not be enough tooth structure to retain a restoration. in such cases, placement of crowns becomes mandatory. This restoration appears similar to a cap and is fitted over the remainder of the natural crown of the tooth. Crowns are often made of gold, porcelain, or porcelain fused to metal.

If the decay has proceeded to involve the dental pulp. the tooth will need to undergo a root canal treatment, which involves removal of the necrotic pulp tissue by thorough debridement, and replaced by Guttapercha points. This leaves the tooth non-vital and devoid of any living tissue. This is then  followed by crown placement, to protect the tooth. may be necessary for the restoration of a tooth.

If the tooth is too far destroyed from the decay process to effectively restore the tooth, tooth extraction is advised. Extractions may also be preferred by patients unable or unwilling to undergo the expense or difficulties in restoring the tooth.